![]() Some patients with septic shock might benefit from drugs such as corticosteroids or activated protein C. Further research is needed to establish which fluids and vasopressors are best. The time-window for interventions is short, and treatment must promptly control the source of infection and restore haemodynamic homoeostasis. The inflammatory response leads to damage to host tissue, and the anti-inflammatory response causes leucocyte reprogramming and changes in immune status. ![]() Proinflammatory mediators that contribute to eradication of invading microorganisms are produced, and anti-inflammatory mediators control this response. ![]() Pathogens, via their microbial-associated molecular patterns, trigger sequential intracellular events in immune cells, epithelium, endothelium, and the neuroendocrine system. In recent years, exciting advances have been made in the understanding of its pathophysiology and treatment. Septic shock, the most severe complication of sepsis, is a deadly disease.
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